But the drug, an antibody that attacks the sticky protein amyloid, does not work as well in people with more advanced disease.
benefitted less from donanemab than participants who do not have the variant, Lilly’s new data showed. The full results also revealed that the drug works far better in patients who start it when they have low levels of another brain protein called tau. Tau is known to increase throughout Alzheimer's progression, although its role in the disease is still poorly understood.
And even after stopping the drug, those who had taken it continued to decline at a slower rate over the course of one year than those who had initially received placebo.
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